100g

L-Glutathione, Reduced, USP Dietary Supplement

L-Histidine

L-Histidine Monohydrochloride, Monohydrate

L-Histidine, FCC

L-Leucine

L-Leucine

L-Leucine, FCC

L-Lysine L-Glutamate Salt

L-Lysine Monohydrochloride

L-Lysine Monohydrochloride, FCC

L-Methionine

L-Ornithine Hydrochloride

L-Phenylalanine

L-Proline

L-Proline, FCC

L-Pyroglutamic Acid

L-Selenomethionine Blend

L-Serine

L-Serine, FCC

L-Threonine, Crystal, FCC

L-Tryptophan

L-Tyrosine

L-Tyrosine Methyl Ester Hydrochloride

L-Tyrosine, FCC

L-Valine, Crystal

L-Valine, Crystal, FCC

Labetalol Hydrochloride, USP

Labetalol Hydrochloride, USP, bioCERTIFIED(TM)

Lactobionic Acid, Reagent

Lactulose, Powder

Lymphocyte activation gene-3 (LAG-3), also known as CD223, is a cell-surface 70kDa molecule belong to Ig superfamily with diverse biologic effects on T cell function. LAG-3 is a CD4 homolog originally cloned in 1990. The gene for LAG-3 lies adjacent to the gene for CD4 on human chromosome 12 (12p13) and is approximately 20% identical to the CD4 gene. human LAG-3 shares 70%, 67%, 76%, and 73% aa sequence identity with mouse, rat, porcine, and bovine LAG-3, respectively. LAG-3 is expressed on B cells, NK cells, tumor-infiltrating lymphocytes, and a subset of T cells. LAG-3 was relatively overexpressed on transgenic T cells rendered anergic in vivo by encounter with cognate self-antigen. LAG-3 negatively regulates murine T cell activation and homeostasis. LAG-3 activates antigen-presenting cells through MHC class II signaling, leading to increased antigen-specific T-cell responses in vivo. Blocking or knocking out LAG-3 in neuronal cultures or in animals mitigated the transmission of α-synuclein between neurons, and dampened accumulation as well as toxic effects of the fibrils on motor function. Anti-LAG3 antibodies are already being tested as cancer treatments, it could also make a useful therapeutic target to treat Parkinson’s and other synucleinopathies.

Lanthanum Acetate, High Purity

Lanthanum Oxide, Reagent

Lauramide

Lauramine Oxide, Solution

Lauric Acid, Reagent

Lauric Anhydride

Lead Dioxide, Powder

Leishman’s Stain

Leucine, USP

Leuco Crystal Violet

Leucovorin Calcium, USP

Levodopa, Powder, USP

Lidocaine

Lidocaine Hydrochloride, Monohydrate, USP

Lidocaine, USP

Light Green SF Yellowish

LIGHT, also known as tumor-necrosis factor (TNF) superfamily member 14 (TNFSF14), is predominantly expressed on activated immune cells and some tumor cells. LIGHT (homologous to lymphotoxin, exhibits inducible expression and competes with Herpes Simplex Virus glycoprotein D for Herpes Virus Entry Mediator, a receptor expressed by T cells), is a protein primarily expressed on activated T cells, activated Natural Killer (NK) cells, and immature dendritic cells (DC). LIGHT can function as both a soluble and cell surface-bound type II membrane protein and must be in its homotrimeric form to interact with its two primary functional receptors: Herpes Virus Entry Mediator (HVEM) and Lymphotoxin-β Receptor (LTβR). LIGHT signaling through these receptors have distinct functions that are cell-type dependent, but interactions with both types of receptors have immune-related implications in tumor biology.

Linoleic Acid Methyl Ester