1mg

Molecular Formula : C15H21NO8

Molecular Formula : C9H9D3ClNO2

Molecular Formula : C23H21ClN4O5S . xHCl

Molecular Formula : 13C8H6O3

Molecular Formula : 13C6H6O3

Molecular Formula : C23 H30 N2 O5

Molecular Formula : C23H27D3N2O4

Molecular Formula : C23 H30 N2 O6

Molecular Formula : C36H45ClN6O12

Molecular Formula : C20 H28 O6

Molecular Formula : C6H12N4Na3O5P

Molecular Formula : C2H4D4NO4P

Molecular Formula : No Data Available

Molecular Formula : C22H22O10

Molecular Formula : C22 H22 O10

Molecular Formula : C16H9D3O5

Molecular Formula : C15 D3 H18 N3 O2

Molecular Formula : C31H48O7

Molecular Formula : C12H20D3NO3

Molecular Formula : C19D4H8F4N2O2

Molecular Formula : C22H19D3O8

Molecular Formula : C16H14BrN5O4S

Molecular Formula : C28H24D5F2N3O

Molecular Formula : C26 H34 N2 O13

Molecular Formula : C14 D7 H13 N2 O2

Molecular Formula : C14 H12 O2

Molecular Formula : C23H29D3N2O4

Molecular Formula : C19H16D4N2O3S

Molecular Formula : C19H17D4ClN2O3S

Molecular Formula : C29 D10 H22 N2 O3

Molecular Formula : C23 2H5 H22 N5 O7 S

Molecular Formula : C23H29N5O8S

Molecular Formula : C29H26D6Cl2N6•4H3PO4

Molecular Formula : C31 H33 K N6 O8 S

Molecular Formula : C24 D10 H21 N5 O3

Molecular Formula : C6 D8 H2 N2 O2

Molecular Formula : C19H12D11Cl2N5O2

Molecular Formula : C19H15D8Cl2N5O2

Molecular Formula : C17 H18 N2 O5 S

Molecular Formula : C17H14D4N2O5S

Molecular Formula : C11 2H6 H12 N4 O2

Molecular Formula : C27 H34 N4 O

Molecular Formula : C17 H31 Cl N2 O5 S . x[HCl]

Molecular Formula : C15 2H3 H10 N3 O4 S

Molecular Formula : C25H17D5FNO3

Molecular Formula : C25H18D5FNNaO4

Molecular Formula : C513C2H1315NO3

Protein Induced by Vitamin K Absence or Antagonist-II (PIVKA-II), also known as Des-γ-carboxy-prothrombin (DCP), is an abnormal form of prothrombin. Normally, the prothrombin’s 10 glutamic acid residues (Glu) in the γ-carboxyglutamic acid (Gla) domain at positions 6, 7, 14, 16, 19, 20,25, 26, 29 and 32 are γ-carboxylated to Gla by vitamin-K dependent γ- glutamyl carboxylase in the liver and then secreted into plasma. In patients with hepatocellular carcinoma (HCC), γ-carboxylation of prothrombin is impaired so that PIVKA-II is formed instead of prothrombin. PIVKA-II is considered as is an efficient biomarker specific for HCC.

Protein Induced by Vitamin K Absence or Antagonist-II (PIVKA-II), also known as Des-γ-carboxy-prothrombin (DCP), is an abnormal form of prothrombin. Normally, the prothrombin’s 10 glutamic acid residues (Glu) in the γ-carboxyglutamic acid (Gla) domain at positions 6, 7, 14, 16, 19, 20,25, 26, 29 and 32 are γ-carboxylated to Gla by vitamin-K dependent γ- glutamyl carboxylase in the liver and then secreted into plasma. In patients with hepatocellular carcinoma (HCC), γ-carboxylation of prothrombin is impaired so that PIVKA-II is formed instead of prothrombin. PIVKA-II is considered as is an efficient biomarker specific for HCC.

Protein Induced by Vitamin K Absence or Antagonist-II (PIVKA-II), also known as Des-γ-carboxy-prothrombin (DCP), is an abnormal form of prothrombin. Normally, the prothrombin’s 10 glutamic acid residues (Glu) in the γ-carboxyglutamic acid (Gla) domain at positions 6, 7, 14, 16, 19, 20,25, 26, 29 and 32 are γ-carboxylated to Gla by vitamin-K dependent γ- glutamyl carboxylase in the liver and then secreted into plasma. In patients with hepatocellular carcinoma (HCC), γ-carboxylation of prothrombin is impaired so that PIVKA-II is formed instead of prothrombin. PIVKA-II is considered as is an efficient biomarker specific for HCC.